The suggestion to limit caffeine to decrease headaches and insomnia is interesting, in view of a recent post about coffee consumption. Do your own research!
Check out a 2007 hcvadvocate.org article by Alan Franciscus, Editor-in-Chief/advocate0907.html#6, about the possible association between coffee consumption and reduction in HCC. Maybe Ms. Porter needs to review her own archives. As I mentioned recently, continued coffee consumption was one of the three post tx "to-dos", along with no alcohol and ultrasounds every six months. Evidently my hepatologist concurs that there may be a connection.
P.S. I'm typing this while enjoying a second cup of coffee, so perhaps some bias is "percolating" through. Lol!
-- Edited by Gator Man on Sunday 3rd of August 2014 04:46:21 PM
Huey said
Aug 3, 2014
I think Alcohol and tobacco are more invasive to the liver Than the estimates indicate, and Those that are 3's and DO NOT drink and use tobacco and other free radical lain substances have a higher degree of Non-Fibrosis cohorts . In my point of view, 3's stand to gain more from not drinking altogether than those in other genotypes.
wmlj1960 said
Aug 2, 2014
Isiscat2011 wrote: Moreover, the article lists stages of HE with early stages defined by symptoms as ubiquitous as as memory loss. The normal aging process will produce such "symptoms."
When they arrive a these statistics, I wander how much research is included for the subjects with history of long term alcoholism, which is the primary contributing cause of cirrhosis in the first place for many cases, and which is a major cause of short term memory loss/confusion Sx's very much resembling HE Sx's.
I bookmarked Mrs Porters site when Malcolm first posted this thread earlier. Now I have that bookmark deleted. I certainly value your opinion after my short time around here.
Thanks Isiscat
Isiscat2011 said
Aug 2, 2014
mallani wrote:
The statement '7 out of 10 cirrhotics develop Hepatic Encephalopathy' is a bit much. HE only develops in decompensated cirrhosis, when ammonia increases.
That statement is ridiculous. Considering that most HepC induced cirrhosis will not progress to decompensation in the span of a lifetime (particularly with treatment of the HepC) is not possible that 7 of 10 cirrhotics will develop HE. Moreover, the article lists stages of HE with early stages defined by symptoms as ubiquitous as as memory loss. The normal aging process will produce such "symptoms."
I have noticed that Lucinda Porter, RN, is often mistaken. While much of the information she relays is reliable and helpful some of her articles leave a great deal to be desired. It appears that Ms. Porter is prone to reading a single study and then reporting it as fact with nary a critical aforethought.
mallani said
Aug 2, 2014
Hi all,
Here's the latest Newsletter.
Some observations:
The suggestion to limit caffeine to decrease headaches and insomnia is interesting, in view of a recent post about coffee consumption. Do your own research!
Mixed cryoglobulinaemia is certainly found in some HCV patients. However, the various symptoms attributed to that are probably due to direct viral infection of various tissues.
The statement '7 out of 10 cirrhotics develop Hepatic Encephalopathy' is a bit much. HE only develops in decompensated cirrhosis, when ammonia increases.
It is well known that Geno 3's develop cirrhosis more quickly, and therefore will have an increased chance of HCC. To say Geno 3's have an 80% increased risk of HCC is incorrect. Cheers.
Check out a 2007 hcvadvocate.org article by Alan Franciscus, Editor-in-Chief/advocate0907.html#6, about the possible association between coffee consumption and reduction in HCC. Maybe Ms. Porter needs to review her own archives. As I mentioned recently, continued coffee consumption was one of the three post tx "to-dos", along with no alcohol and ultrasounds every six months. Evidently my hepatologist concurs that there may be a connection.
http://www.hcvadvocate.org/news/newsLetter/2007/advocate0907.html#1
-John
P.S. I'm typing this while enjoying a second cup of coffee, so perhaps some bias is "percolating" through. Lol!
-- Edited by Gator Man on Sunday 3rd of August 2014 04:46:21 PM
I think Alcohol and tobacco are more invasive to the liver Than the estimates indicate, and Those that are 3's and DO NOT drink and use tobacco and other free radical lain substances have a higher degree of Non-Fibrosis cohorts . In my point of view, 3's stand to gain more from not drinking altogether than those in other genotypes.
When they arrive a these statistics, I wander how much research is included for the subjects with history of long term alcoholism, which is the primary contributing cause of cirrhosis in the first place for many cases, and which is a major cause of short term memory loss/confusion Sx's very much resembling HE Sx's.
I bookmarked Mrs Porters site when Malcolm first posted this thread earlier. Now I have that bookmark deleted. I certainly value your opinion after my short time around here.
Thanks Isiscat
That statement is ridiculous. Considering that most HepC induced cirrhosis will not progress to decompensation in the span of a lifetime (particularly with treatment of the HepC) is not possible that 7 of 10 cirrhotics will develop HE. Moreover, the article lists stages of HE with early stages defined by symptoms as ubiquitous as as memory loss. The normal aging process will produce such "symptoms."
I have noticed that Lucinda Porter, RN, is often mistaken. While much of the information she relays is reliable and helpful some of her articles leave a great deal to be desired. It appears that Ms. Porter is prone to reading a single study and then reporting it as fact with nary a critical aforethought.
Hi all,
Here's the latest Newsletter.
Some observations:
The suggestion to limit caffeine to decrease headaches and insomnia is interesting, in view of a recent post about coffee consumption. Do your own research!
Mixed cryoglobulinaemia is certainly found in some HCV patients. However, the various symptoms attributed to that are probably due to direct viral infection of various tissues.
The statement '7 out of 10 cirrhotics develop Hepatic Encephalopathy' is a bit much. HE only develops in decompensated cirrhosis, when ammonia increases.
It is well known that Geno 3's develop cirrhosis more quickly, and therefore will have an increased chance of HCC. To say Geno 3's have an 80% increased risk of HCC is incorrect. Cheers.
http://www.hcvadvocate.org/news/newsLetter/2014/advocate0814.html
-- Edited by mallani on Sunday 3rd of August 2014 12:25:11 AM