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Hep C Discussion Forum -> Hep C News -> August Edition- HCV Advocate newsletter
Post Info TOPIC: August Edition- HCV Advocate newsletter
Gator Man


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August Edition- HCV Advocate newsletter
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mallani wrote:

Hi all,

Here's the latest Newsletter.

Some observations:

The suggestion to limit caffeine to decrease headaches and insomnia is interesting, in view of a recent post about coffee consumption. Do your own research!

http://www.hcvadvocate.org/news/newsLetter/2014/advocate0814.html

 

Check out a 2007 hcvadvocate.org article by Alan Franciscus, Editor-in-Chief/advocate0907.html#6, about the possible association between coffee consumption and reduction in HCC. Maybe Ms. Porter needs to review her own archives. As I mentioned recently, continued coffee consumption was one of the three post tx "to-dos", along with no alcohol and ultrasounds every six months. Evidently my hepatologist concurs that there may be a connection.

http://www.hcvadvocate.org/news/newsLetter/2007/advocate0907.html#1

-John

P.S. I'm typing this while enjoying a second cup of coffee, so perhaps some bias is "percolating" through. Lol!

 



-- Edited by Gator Man on Sunday 3rd of August 2014 04:46:21 PM

__________________

Geno 1b, compensated cirrhotic, 54 yo, prior null responder. Pre tx VL approx 595,000, tx with Sovaldi/Olysio (no Riba) started 1/8/14. VL 40 @ 2 weeks, UND @ 4 weeks. Still UND @ EOT + 1 year.

Gator Man SVR12, Dragon 0, Final Score.
Huey


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I think Alcohol and tobacco are more invasive to the liver Than the estimates  indicate, and Those that are 3's and DO NOT drink and use tobacco and other free radical lain substances have a higher degree of Non-Fibrosis cohorts . In my point of view, 3's stand to gain more from not drinking altogether than those in other genotypes.



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  HCV Genotype 3a , now Psot-Tx was on S/riba. First VL was 5.8 mil on 7-5-13 then "und" at 3.8 weeks. 06/13/14 still und. off meds 3 days back on 7/29 Last pill 08/10/14 SVR+4

 
wmlj1960


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Isiscat2011 wrote:
  Moreover, the article lists stages of HE with early stages defined by symptoms as ubiquitous as as memory loss.  The normal aging process will produce such "symptoms."    

 

When they arrive a these statistics, I wander how much research is included for the subjects with history of long term alcoholism, which is the primary contributing cause of cirrhosis in the first place for many cases, and which is a major cause of short term memory loss/confusion Sx's very much resembling HE Sx's.

I bookmarked Mrs Porters site when Malcolm first posted this thread earlier. Now I have that bookmark deleted. I certainly value your opinion after my short time around here.

Thanks Isiscat smile

 



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60 yo, geno 1a, Dx 1994 HCV-HIV co-inf, Dx 2013 decompensated cirrhosis
Tx #1 - 24wks Sov+Riba /SOT 7-24-2014/UND@EOT/DETECTED@EOT+16 wks
Tx #2 - 24wks Harvoni /SOT 7-25-2015/UND@EOT,+12,+24,+52 = SVR

Mike

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Isiscat2011


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mallani wrote:

The statement '7 out of 10 cirrhotics develop Hepatic Encephalopathy' is a bit much. HE only develops in decompensated cirrhosis, when ammonia increases.

That statement is ridiculous.  Considering that most HepC induced cirrhosis will not progress to decompensation in the span of a lifetime (particularly with treatment of the HepC) is not possible that 7 of 10 cirrhotics will develop HE.   Moreover, the article lists stages of HE with early stages defined by symptoms as ubiquitous as as memory loss.  The normal aging process will produce such "symptoms."    

I have noticed that Lucinda Porter, RN, is often mistaken.  While much of the information she relays is reliable and helpful some of her articles leave a great deal to be desired.  It appears that Ms. Porter is prone to reading a single study and then reporting it as fact with nary a critical aforethought.  



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Diagnosed in 2011, Incivek triple in 2011, tx discontinued, Genotype 1a, CT, VL 7mill, cirrhosis dx in 2012, age 67, waiting for new DAAs.
mallani


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Hi all,

Here's the latest Newsletter.

Some observations:

The suggestion to limit caffeine to decrease headaches and insomnia is interesting, in view of a recent post about coffee consumption. Do your own research!

Mixed cryoglobulinaemia is certainly found in some HCV patients. However, the various symptoms attributed to that are probably due to direct viral infection of various tissues.

The statement '7 out of 10 cirrhotics develop Hepatic Encephalopathy' is a bit much. HE only develops in decompensated cirrhosis, when ammonia increases.

It is well known that Geno 3's develop cirrhosis more quickly, and therefore will have an increased chance of HCC. To say Geno 3's have an 80% increased risk of HCC is incorrect. Cheers.

http://www.hcvadvocate.org/news/newsLetter/2014/advocate0814.html



-- Edited by mallani on Sunday 3rd of August 2014 12:25:11 AM

__________________

Geno 1b, IL28B CT,  x3 prior relapser,  ex-cirrhotic, 75 yo, did 48 weeks with Victrelis/Peg./Riba.  VL 1.28m at start, UNDET. at 8 ,12 ,16 ,24 ,30  and 48 weeks.  EOT 15 Feb 2013 , UNDET. at EOT + 28 weeks. SVR!  Still Undet. at EOT +5 years

Malcolm
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